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Whole body rejuvination

What is Telomind?

The name TeloMind™ is derived from "Telomeres" and "Mind" because of the positive benefits it has on both.

Humans have 23 pairs of chromosomes which are made up of all our genes. Telomeres are the sections of DNA found at the end of each chromosome. These are like little caps which serve to protect the DNA in the chromosome. They are often compared to the plastic protective tips in shoelaces that protect a shoelace from unravelling. In this same way, telomeres play an important role in protecting the DNA and keeping it stable.

As we get older, our telomeres get shorter and shorter. This is a normal part of the aging process but it happens at different rates in different people.

With every cell division, inefficiencies from reactive oxygen species damage cause a small part of the telomeres to fail to copy properly and this causes them to gradually shrink. Shorter telomeres allow your DNA to become unstable and prone to damage which creates disease.

The shorter your telomeres, the older you are from biological perspective. The opposite is also true. The longer your telomeres remain, the younger your cells are biologically; and the better your health.

The second part of the name represents the difference TeloMind™ makes to your mind, through managing the stress hormone cortisol. This enables you to benefit from a positive mood, deep restful sleep, focus and concentration.

People who take Telomind on an ongoing basis can significantly reduce their biological age within just 6 months or so.

As well as pure Norwegian Young Tissue Extract (YTE) TeloMind also contains the important herb Rhodiola rosea, which has traditional usage as an anti-fatigue agent, an adaptogen, and to improve cognitive functioning. Rhodiola rosea is also potentially highly neuroprotective against toxins. In the brain, Rhodiola rosea appears to be highly serotonergic (increases serotonin) and reduces corticosteroids. Rhodiola may also promote longevity, with preliminary evidence suggesting up to a 20% increase in lifespan.

All the cells in our body contain tiny clocks called telomeres that can determine how long they will live. Telomeres are little caps at the end of chromosomes that prevent loss or injury to genetic information during cell division. Each time a cell divides, part of the telomere is lost and it becomes shorter. When a telomere eventually disappears because of repeated cell divisions, chromosomal damage prevents the cell from accurately reproducing itself. Many believe that telomere destruction and reconstruction is related to the balance between aging and cancer and explains why cancer is more common in the elderly.

Cells with long telomeres live longer. Short telomeres have been linked to a wide range of human diseases, including coronary heart disease, osteoporosis, and HIV infection. Shortening of telomeres is prevented or reduced by telomerase, which has been shown to keep immune cells young by preserving their length and ability to continue to divide and reproduce accurate replicas.

UCLA researchers recently confirmed prior reports that people subjected to chronic stress tended to have shorter telomeres. They have now uncovered a mechanism that explains how stress causes telomere shortening, which could lead to breaking the well-known links between stress and heart disease, as well as accelerated aging.

Chronic stress results in increased secretion of cortisol that causes a rise in blood sugar and blood pressure and reduces inflammation and immune system resistance to infection. However, this new study shows that cortisol also suppresses telomerase activation in immune system cells so that telomeres are no longer protected during cell division and become progressively shorter. This leads to early cell aging and distorted replicas of the original cell that could lead to cancer and other diseases. As the lead author noted in an interview, “We are testing therapeutic ways of enhancing telomerase levels to help the immune system ward off cortisol’s effect. If we’re successful, one day a pill may exist to strengthen the immune system’s ability to weather chronic emotional stress.”"

This study was approved by the UCLA Institutional Review Board:

Choi J. Fauce SR, Effros RB. Reduced telomerase activity in human T lymphocytes exposed to cortisol. Brain Behavior and Immunity May 2008; 22:600-605 - -

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